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DOI 10.2214/ajr.168.5.9129430

Медицинская библиотека · AJR. American journal of roentgenology · 1997

Deep fascial hyperintensity in soft-tissue abnormalities as revealed by T2-weighted MR imaging.

Loh N N, Ch'en I Y, Cheung L P, Li K C

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Аннотация

OBJECTIVE

The purpose of this study was to determine whether recently described criteria, including hyperintense T2-weighted signal or other abnormalities revealed by MR imaging within deep fascial planes, are specific for necrotizing soft-tissue infections.

MATERIALS AND

METHODS

We reviewed 22 MR imaging examinations that revealed abnormally high signal intensity within deep fascial planes on T2-weighted images. Twenty-one of the patients had clinical diagnoses other than necrotizing soft-tissue infection, including nonnecrotizing cellulitis (n = 4), abscess without evidence of necrotizing fasciitis (n = 5), and cellulitis with accompanying vascular thrombosis (n = 2). MR imaging was performed using T1-weighted spin-echo (range of TRs/range of TEs, 300-800/9-30) and fat-saturated T2-weighted fast spin-echo (3000-5000/76-108) sequences. Gadolinium-enhanced T1-weighted spin-echo MR images with fat saturation were obtained for 14 patients. Two reviewers, unaware of clinical diagnoses, evaluated each study for abnormalities in superficial and deep soft tissues and submitted a consensus diagnosis.

RESULTS

Using only the described MR imaging criteria, we interpreted all cases as necrotizing cellulitis, including the cases of 21 patients who had nonnecrotizing conditions.

CONCLUSION

Hyperintense T2-weighted signal within deep fascial planes and muscle, with or without contrast enhancement, is not specific for necrotizing soft-tissue infection. A variety of conditions exhibited similar findings and were indistinguishable from necrotizing soft-tissue infection.

Для цитирования: Loh N N, Ch'en I Y, Cheung L P, Li K C. Deep fascial hyperintensity in soft-tissue abnormalities as revealed by T2-weighted MR imaging.. AJR. American journal of roentgenology. 1997. DOI: 10.2214/ajr.168.5.9129430.

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