Cell · 2008
Transcription factor E2-2 is an essential and specific regulator of plasmacytoid dendritic cell development.
СтатьяПоддержка NIH: внешняя программаПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Cisse Babacar, Caton Michele L, Lehner Manfred, Maeda Takahiro, Scheu Stefanie, Locksley Richard, Holmberg Dan, Zweier Christiane, den Hollander Nicolette S, Kant Sarina G, Holter Wolfgang, Rauch Anita, Zhuang Yuan, Reizis Boris
Аннотация
Plasmacytoid dendritic cells (PDCs) represent a unique immune cell type specialized in type I interferon (IFN) secretion in response to viral nucleic acids. The molecular control of PDC lineage specification has been poorly understood. We report that basic helix-loop-helix transcription factor (E protein) E2-2/Tcf4 is preferentially expressed in murine and human PDCs. Constitutive or inducible deletion of murine E2-2 blocked the development of PDCs but not of other lineages and abolished IFN response to unmethylated DNA. Moreover, E2-2 haploinsufficiency in mice and in human Pitt-Hopkins syndrome patients was associated with aberrant expression profile and impaired IFN response of the PDC. E2-2 directly activated multiple PDC-enriched genes, including transcription factors involved in PDC development (SpiB, Irf8) and function (Irf7). These results identify E2-2 as a specific transcriptional regulator of the PDC lineage in mice and humans and reveal a key function of E proteins in the innate immune system.
Предметные рубрики MeSH
AdolescentAnimalsBasic Helix-Loop-Helix Leucine Zipper Transcription FactorsChildChild, PreschoolDNA-Binding ProteinsDendritic CellsHumansHyperventilationImmunity, InnateIntellectual DisabilityInterferonsMiceNerve Tissue ProteinsSyndromeTCF Transcription FactorsTranscription Factor 4Transcription Factor 7-Like 2 ProteinTranscription Factors