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DOI 10.1007/s11064-013-1011-6

Медицинская библиотека · Neurochemical research · 2013

Changes in Ataxin-10 expression after sciatic nerve crush in adult rats.

Cheng Xinghai, Gan Lin, Zhao Jing, Chen Minhao, Liu Yang, Wang Youhua

СтатьяПоддержка исследования: вне правительства СШАПеревод в очереди

Аннотация

Ataxin-10 is a cytoplasmic protein that belongs to the family of armadillo repeat proteins and the ataxin proteins are ubiquitously expressed in nervous tissue. A loss of Ataxin-10 in primary neuronal cells causes increased apoptosis of cerebellar neurons. Knockdown of ATXN10 with siRNA in HeLa cells results in cytokinesis defects-multinucleation. Because of the essential role of Ataxin-10 in nervous system and cellular cytokinesis, we investigated the spatiotemporal expression of Ataxin-10 in a rat sciatic nerve crush (SNC) model. After never injury, we observed that Ataxin-10 had a significant up-regulation from 3d, peaked at day 5 and then gradually decreased to the normal level at 4 weeks. At its peak expression, Ataxin-10 expressed mainly in Schwann cells and macrophages of the distal sciatic nerve segment from injury, but had few co-localizations in axons. Besides, the peak expression of Ataxin-10 was in parallel with proliferating cell nuclear antigen (PCNA), and Ataxin-10 co-labeled with PCNA. Thus, all of our findings suggested that Ataxin-10 may be involved in the pathophysiology of sciatic nerve after SNC.

Для цитирования: Cheng Xinghai, Gan Lin, Zhao Jing, Chen Minhao, Liu Yang, Wang Youhua. Changes in Ataxin-10 expression after sciatic nerve crush in adult rats.. Neurochemical research. 2013. DOI: 10.1007/s11064-013-1011-6.

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