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Brain : a journal of neurology · 2026

Variants in DENND2B are associated with vulnerability for neurodevelopmental impairment, psychosis and catatonia.

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Murthy Harsha, Hoang Ny, Stark Jamie C, Cui Sunny, Pannia Emanuela, Tsoi Chung Ting, Harris Simon, Ceolin C'airah, Verhaeghe Lauren, Scholten Sydney, Baribeau Danielle, Summers Jane, Costain Gregory, Selvanayagam Thanuja, Howe Jennifer L, Lewis M E Suzanne, Brunet Theresa, Rieger Susanne, Rosenfeld Jill A, Craigen William J, Burrage Lindsay C, Christie Michelle R, Baldwin Deborah, Wentzensen Ingrid M, Keren Boris, Cogne Benjamin, Isidor Bertrand, Afenjar Alexandra, Elshafie Reem M, Bastaki Laila, Alkanderi Sumaya, Myers Kenneth A, Demarest Scott, Angione Katie, Abbott Megan, Campeau Philippe M, Dowling James J, Mendoza-Londono Roberto, Scherer Stephen W, Deshwar Ashish R, Vorstman Jacob

Аннотация

DENND2B is a DENN (differentially expressed in normal and neoplastic cells) domain-containing protein that has important roles in regulating the cell cycle, cell division and ciliogenesis, but to date has not been associated with any human disease. Here, we report on 11 individuals with monoallelic variants in DENND2B with a shared constellation of features and perform in silico and in vivo zebrafish modelling of the DENND2B variants identified in these patients. Features shared among these patients include developmental delay, intellectual disability and psychiatric/behavioural concerns, and episodes of psychosis and/or catatonia. Additional features common to our cohort include epilepsy, muscle weakness/hypotonia and a wide range of congenital anomalies across different organ systems. Identified patient variants affect well-conserved amino acids and are predicted to be deleterious to DENND2B function by in silico prediction algorithms and structural modelling. Nine of the 10 observed patient variants were modelled in zebrafish and confirmed to result in loss of DENND2B function. Altogether, these findings suggest that monoallelic loss-of-function variants in DENND2B cause a novel autosomal dominant neurodevelopmental disorder with variable vulnerability to psychosis and/or catatonia.

Предметные рубрики MeSH

ИП Урсолов Владлен Сергеевич

ИНН 500118482352 · ОГРНИП 314500117700049

Реквизиты и контакты