Journal of epidemiology and community health · 2010
Air pollution and mortality in New Zealand: cohort study.
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Hales Simon, Blakely Tony, Woodward Alistair
Аннотация
BACKGROUND: Few cohort studies of the health effects of urban air pollution have been published. There is evidence, most consistently in studies with individual measurement of social factors, that more deprived populations are particularly sensitive to air pollution effects.
METHODS: Records from the 1996 New Zealand census were anonymously and probabilistically linked to mortality data, creating a cohort study of the New Zealand population followed up for 3 years. There were 1.06 million adults living in urban areas for which data were available on all covariates. Estimates of exposure to air pollution (measured as particulate matter with an aerodynamic diameter less than 10 μm, PM(10)) were available for census area units from a previous land use regression study. Logistic regression analyses were conducted to investigate associations between cause-specific mortality rates and average exposure to PM(10) in urban areas, with control for confounding by age, sex, ethnicity, social deprivation, income, education, smoking history and ambient temperature.
RESULTS: The odds of all-cause mortality in adults (aged 30-74 years at census) increased by 7% per 10 μg/m(3) increase in average PM(10) exposure (95% CI 3% to 10%) and 20% per 10 μg/m(3) among Maori, but with wide CI (7% to 33%). Associations were stronger for respiratory and lung cancer deaths.
CONCLUSIONS: An association of PM(10) with mortality is reported in a country with relatively low levels of air pollution. The major limitation of the study is the probable misclassification of PM(10) exposure. On balance, this means the strength of association was probably underestimated. The apparently greater association among Maori might be due to different levels of co-morbidity.
METHODS: Records from the 1996 New Zealand census were anonymously and probabilistically linked to mortality data, creating a cohort study of the New Zealand population followed up for 3 years. There were 1.06 million adults living in urban areas for which data were available on all covariates. Estimates of exposure to air pollution (measured as particulate matter with an aerodynamic diameter less than 10 μm, PM(10)) were available for census area units from a previous land use regression study. Logistic regression analyses were conducted to investigate associations between cause-specific mortality rates and average exposure to PM(10) in urban areas, with control for confounding by age, sex, ethnicity, social deprivation, income, education, smoking history and ambient temperature.
RESULTS: The odds of all-cause mortality in adults (aged 30-74 years at census) increased by 7% per 10 μg/m(3) increase in average PM(10) exposure (95% CI 3% to 10%) and 20% per 10 μg/m(3) among Maori, but with wide CI (7% to 33%). Associations were stronger for respiratory and lung cancer deaths.
CONCLUSIONS: An association of PM(10) with mortality is reported in a country with relatively low levels of air pollution. The major limitation of the study is the probable misclassification of PM(10) exposure. On balance, this means the strength of association was probably underestimated. The apparently greater association among Maori might be due to different levels of co-morbidity.