Revista de neurologia · 2005
[Developmental stuttering and acquired stuttering: resemblances and differences].
Аннотация на английскомСтатьяНазвание и аннотация: Перевод пока не подготовлен
Обработка статьи · этапы, ошибки и управление
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Manaut-Gil E
Аннотация
INTRODUCTION: In this study the authors analyse, clinically, dysphemia (DP) and acquired stuttering (AS). AIMS. The aim of this study was to evaluate whether AS is a variant of DP or whether it is an entity that shares a common element: stuttered speech.
PATIENTS AND METHODS: The authors studied 13 patients with AS and 36 with DP. In addition to the clinical evaluation, electroencephalogram (EEG) and cranial computerised axial tomography (CAT) scans were performed, with a special interest in secondary symptoms/signs, laterality profiles and pathological, personal and familial history.
RESULTS: There was a notable predominance in males in both groups. AS began either in infancy or from any other age; DP only started in infancy. The most frequent organic pathology, for both DP and AS, was a severe traumatic brain injury, followed by cerebral anoxia/hypoxia, cerebrovascular accident (CVA) and others. One important element in both groups was the presence of stuttering and high percentages of left-handedness in the families. In AS, all patients were right-handed. None of the patients who experienced the onset of AS in infancy improved/yielded during adolescence.
CONCLUSIONS: Both DP and AS are cases of "neurogenic" stuttering because they display organic and/or functional pathologies in the two groups, which invalidates the term "developmental", since AS also occurred in infancy. DP and AS have an element in common: they both share a genetic predisposition on which the organic/functional pathology then gives rise to the clinical symptoms, although this does not account for the absence of tics in AS.
PATIENTS AND METHODS: The authors studied 13 patients with AS and 36 with DP. In addition to the clinical evaluation, electroencephalogram (EEG) and cranial computerised axial tomography (CAT) scans were performed, with a special interest in secondary symptoms/signs, laterality profiles and pathological, personal and familial history.
RESULTS: There was a notable predominance in males in both groups. AS began either in infancy or from any other age; DP only started in infancy. The most frequent organic pathology, for both DP and AS, was a severe traumatic brain injury, followed by cerebral anoxia/hypoxia, cerebrovascular accident (CVA) and others. One important element in both groups was the presence of stuttering and high percentages of left-handedness in the families. In AS, all patients were right-handed. None of the patients who experienced the onset of AS in infancy improved/yielded during adolescence.
CONCLUSIONS: Both DP and AS are cases of "neurogenic" stuttering because they display organic and/or functional pathologies in the two groups, which invalidates the term "developmental", since AS also occurred in infancy. DP and AS have an element in common: they both share a genetic predisposition on which the organic/functional pathology then gives rise to the clinical symptoms, although this does not account for the absence of tics in AS.