Scientific reports · 2019
Fat cadherins in mouse models of degenerative ataxias.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод пока не подготовлен
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Baron Olga, Grieshober Denise, Dias Catarina, Fanto Manolis
Аннотация
Autophagy is a lysosomal degradation pathway that plays an essential role in neuronal homeostasis and is perturbed in many neurological diseases. Transcriptional downregulation of fat was previously observed in a Drosophila model of the polyglutamine disease Dentatorubral-pallidoluysian atrophy (DRPLA) and this was shown to be partially responsible for autophagy defects and neurodegeneration. However, it is still unclear whether a downregulation of mammalian Fat orthologues is associated with neurodegeneration in mice. We hereby show that all four Fat orthologues are transcriptionally downregulated in the cerebellum in a mouse model of DRPLA. To elucidate the possible roles of single Fat genes, this study concentrates on Fat3. This fat homologue is shown to be the most widely expressed in the brain. Conditional knockout (KO) of Fat3 in brains of adult mice was attempted using the inducible Thy1Cre(ERT2) SLICK H line. Behavioral and biochemical analysis revealed that mice with conditional KO of Fat3 in the brain display no abnormalities. This may be ascribed either to the limited efficiency of the KO strategy pursued or to the lack of effect of Fat3 KO on autophagy.
Предметные рубрики MeSH
AnimalsAtaxiaAutophagyBrainCadherinsCerebellumDisease Models, AnimalDown-RegulationGenes, SyntheticHeredodegenerative Disorders, Nervous SystemHippo Signaling PathwayIntegrasesMiceMice, Inbred StrainsMice, KnockoutMyoclonic Epilepsies, ProgressiveNerve Tissue ProteinsOlfactory BulbOrgan SpecificityPromoter Regions, GeneticProtein Serine-Threonine KinasesRecombinant Fusion ProteinsThy-1 Antigens