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Brain : a journal of neurology · 2006

MFN2 mutation distribution and genotype/phenotype correlation in Charcot-Marie-Tooth type 2.

СтатьяМногоцентровое исследованиеПоддержка NIH: внешняя программаПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод пока не подготовлен
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Verhoeven Kristien, Claeys Kristl G, Züchner Stephan, Schröder J Michael, Weis Joachim, Ceuterick Chantal, Jordanova Albena, Nelis Eva, De Vriendt Els, Van Hul Matthias, Seeman Pavel, Mazanec Radim, Saifi Gulam Mustafa, Szigeti Kinga, Mancias Pedro, Butler Ian J, Kochanski Andrzej, Ryniewicz Barbara, De Bleecker Jan, Van den Bergh Peter, Verellen Christine, Van Coster Rudy, Goemans Nathalie, Auer-Grumbach Michaela, Robberecht Wim, Milic Rasic Vedrana, Nevo Yoram, Tournev Ivajlo, Guergueltcheva Velina, Roelens Filip, Vieregge Peter, Vinci Paolo, Moreno Maria Teresa, Christen H-J, Shy Michael E, Lupski James R, Vance Jeffery M, De Jonghe Peter, Timmerman Vincent

Аннотация

Mutations in mitofusin 2 (MFN2) have been reported in Charcot-Marie-Tooth type 2 (CMT2) families. To study the distribution of mutations in MFN2 we screened 323 families and isolated patients with distinct CMT phenotypes. In 29 probands, we identified 22 distinct MFN2 mutations, and 14 of these mutations have not been reported before. All mutations were located in the cytoplasmic domains of the MFN2 protein. Patients presented with a classical but rather severe CMT phenotype, since 28% of them were wheelchair-dependent. Some had additional features as optic atrophy. Most patients had an early onset and severe disease status, whereas a smaller group experienced a later onset and milder disease course. Electrophysiological data showed in the majority of patients normal to slightly reduced nerve conduction velocities with often severely reduced amplitudes of the compound motor and sensory nerve action potentials. Examination of sural nerve specimens showed loss of large myelinated fibres and degenerative mitochondrial changes. In patients with a documented family history of CMT2 the frequency of MFN2 mutations was 33% indicating that MFN2 mutations are a major cause in this population.

Предметные рубрики MeSH

ИП Урсолов Владлен Сергеевич

ИНН 500118482352 · ОГРНИП 314500117700049

Реквизиты и контакты