Stem cells and development · 2012
Multiple low-dose infusions of human umbilical cord blood cells improve cognitive impairments and reduce amyloid-β-associated neuropathology in Alzheimer mice.
СтатьяПоддержка NIH: внешняя программаНазвание и аннотация: Перевод пока не подготовлен
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Darlington Donna, Deng Juan, Giunta Brian, Hou Huayan, Sanberg Cyndy D, Kuzmin-Nichols Nicole, Zhou Hua-Dong, Mori Takashi, Ehrhart Jared, Sanberg Paul R, Tan Jun
Аннотация
Alzheimer's disease (AD) is the most common progressive age-related dementia in the elderly and the fourth major cause of disability and mortality in that population. The disease is pathologically characterized by deposition of β-amyloid plaques neurofibrillary tangles in the brain. Current strategies for the treatment of AD are symptomatic only. As such, they are less than efficacious in terms of significantly slowing or halting the underlying pathophysiological progression of the disease. Modulation by cell therapy may be new promising disease-modifying therapy. Recently, we showed reduction in amyloid-β (Aβ) levels/β-amyloid plaques and associated astrocytosis following low-dose infusions of mononuclear human umbilical cord blood cells (HUCBCs). Our current study extended our previous findings by examining cognition via (1) the rotarod test, (2) a 2-day version of the radial-arm water maze test, and (3) a subsequent observation in an open pool platform test to characterize the effects of monthly peripheral HUCBC infusion (1×10(6) cells/μL) into the transgenic PSAPP mouse model of cerebral amyloidosis (bearing mutant human APP and presenilin-1 transgenes) from 6 to 12 months of age. We show that HUCBC therapy correlates with decreased (1) cognitive impairment, (2) Aβ levels/β-amyloid plaques, (3) amyloidogenic APP processing, and (4) reactive microgliosis after a treatment of 6 or 10 months. As such, this report lays the groundwork for an HUCBC therapy as potentially novel alternative to oppose AD at the disease-modifying level.
Предметные рубрики MeSH
Alzheimer DiseaseAmyloid beta-Protein PrecursorAnimalsBrainCells, CulturedCognitionCord Blood Stem Cell TransplantationFemaleHumansInfusions, ParenteralLeukocyte Common AntigensMaleMaze LearningMemory, Short-TermMiceMice, TransgenicMicrogliaMotor SkillsPlaque, AmyloidRecovery of FunctionRotarod Performance Test