Atherosclerosis · 1996
Apolipoprotein E1-Hammersmith (Lys146-->Asn;Arg147-->Trp), due to a dinucleotide substitution, is associated with early manifestation of dominant type III hyperlipoproteinaemia.
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Hoffer M J, Niththyananthan S, Naoumova R P, Kibirige M S, Frants R R, Havekes L M, Thompson G R
Аннотация
Apolipoprotein E (apoE) is one of the major protein constituents of chylomicron and very low density lipoprotein (VLDL) remnants and plays a central role as a ligand in the receptor-mediated uptake of these particles by the liver. Here we describe a new variant of apoE, apoE1-Hammersmith, which is associated with dominantly expressed type III hyperlipidaemia. The propositus, aged 26, developed tubero-eruptive xanthomas at the age of 3, her daughter developed similar lesions at age 7 but her son, aged 3, shows no clinical abnormality so far. All three cases had an apoE3E1 phenotype and a broad beta band on lipoprotein electrophoresis. Cysteamine modification resulted in a shift of apoE1 to the apoE2 isoform position, indicating that the mutation leading to apoE1-Hammersmith occurred on an apoE3 background. ApoE genotyping confirmed these results. Sequence analysis of DNA of the propositus was performed for exons 3 and 4 and revealed a dinucleotide substitution causing two amino acid changes at adjacent positions (Lys146-->Asn) and (Arg147-->Trp).
Предметные рубрики MeSH
AdultAnticholesteremic AgentsApolipoproteins EChildChild, PreschoolCholesterolCholestyramine ResinCysteamineDNADinucleotide RepeatsElectrophoresisExonsFemaleFenofibrateGenotypeHumansHyperlipoproteinemia Type IIIHypolipidemic AgentsImmunoblottingMaleNuclear FamilyPhenotypePoint MutationRadiation-Protective AgentsTriglycerides