Scientific reports · 2016
CADM1 inhibits squamous cell carcinoma progression by reducing STAT3 activity.
СтатьяПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод пока не подготовлен
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Vallath Sabari, Sage Elizabeth K, Kolluri Krishna K, Lourenco Sofia N, Teixeira Vitor S, Chimalapati Suneeta, George P Jeremy, Janes Sam M, Giangreco Adam
Аннотация
Although squamous cell carcinomas (SqCCs) of the lungs, head and neck, oesophagus, and cervix account for up to 30% of cancer deaths, the mechanisms that regulate disease progression remain incompletely understood. Here, we use gene transduction and human tumor xenograft assays to establish that the tumour suppressor Cell adhesion molecule 1 (CADM1) inhibits SqCC proliferation and invasion, processes fundamental to disease progression. We determine that the extracellular domain of CADM1 mediates these effects by forming a complex with HER2 and integrin α6β4 at the cell surface that disrupts downstream STAT3 activity. We subsequently show that treating CADM1 null tumours with the JAK/STAT inhibitor ruxolitinib mimics CADM1 gene restoration in preventing SqCC growth and metastases. Overall, this study identifies a novel mechanism by which CADM1 prevents SqCC progression and suggests that screening tumours for loss of CADM1 expression will help identify those patients most likely to benefit from JAK/STAT targeted chemotherapies.
Предметные рубрики MeSH
AnimalsCarcinoma, Squamous CellCell Adhesion Molecule-1Cell Adhesion MoleculesCell Line, TumorCell MovementCell ProliferationDisease ProgressionFemaleGene Expression ProfilingHumansImmunoglobulinsIntegrin alpha6beta4Lung NeoplasmsMembrane ProteinsMiceNeoplasm InvasivenessNeoplasm MetastasisNeoplasm TransplantationNitrilesPyrazolesPyrimidinesErb-b2 Receptor Tyrosine KinasesSTAT3 Transcription FactorUterine Cervical Neoplasms