Clinical pharmacokinetics · 2016
Clinical Pharmacokinetics and Pharmacodynamics of Bosutinib.
СтатьяОбзорНазвание и аннотация: Перевод пока не подготовлен
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Abbas Richat, Hsyu Poe-Hirr
Аннотация
Chronic myeloid leukemia (CML) is a clonal myeloproliferative stem cell disorder. Bosutinib is an oral, once-daily SRC/ABL tyrosine kinase inhibitor with very potent inhibitory activity. Bosutinib is effective against all phases of intolerant or resistant Philadelphia chromosome-positive CML that do not harbor the T315I or V299LABL kinase domain mutations. Peak plasma concentrations of bosutinib occur at 4-6 h following oral administration, and dose-proportional increases in exposure are observed at doses ranging from 200 to 800 mg. Absorption of bosutinib increases with food. Bosutinib is distributed extensively into the tissues. It is highly plasma protein bound (94 %) and is primarily metabolized in the liver by cytochrome P450 3A4. Bosutinib is well tolerated overall and has a unique but manageable toxicity profile. This article provides a review of the available clinical pharmacokinetic, pharmacodynamic, and drug-drug interaction data on bosutinib in healthy subjects, patients with CML, and special populations.
Предметные рубрики MeSH
Aniline CompoundsAntineoplastic AgentsArea Under CurveAsian PeopleClinical Trials as TopicCytochrome P-450 CYP3ADose-Response Relationship, DrugDrug InteractionsElectrocardiographyHalf-LifeHumansIntestinal AbsorptionLeukemia, Myelogenous, Chronic, BCR-ABL PositiveLiver FailureMetabolic Clearance RateNitrilesProtein BindingProtein Kinase InhibitorsQuinolinesRenal Insufficiency