The Journal of experimental medicine · 2026
Bile acid retention in efferocytic macrophages shapes their inflammatory status during cholangitis.
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Al Jawazneh Amirah, Liebold Imke, Leyk Stephanie, Lanzloth Clarissa, Brackrock Vera, López-López Tamara, Graute Sebastian, Aranda-Pardos Irene, Hamley Madeleine, Bahn Jonas, Heeren Joerg, Von Felden Johann, Lohse Ansgar W, Schramm Christoph, Schwinge Dorothee, Sáez Pablo J, A-Gonzalez Noelia, Huber Samuel, Jacobs Thomas, Adlung Lorenz, Worthmann Anna, Bosurgi Lidia
Аннотация
The clearance of apoptotic cells by phagocytes is crucial for restoring tissue balance after injury. In autoimmune liver diseases like primary sclerosing cholangitis, cell death is thought to result from accumulation of toxic bile acids within parenchymal cells. Whether, in this context, bile acid-loaded dying cells impact the efficiency of phagocytic macrophages in restoring tissue balance remains unknown. Here, we demonstrate that in a murine model of cholangitis, bile acids accumulate in a subpopulation of efferocytic macrophages with pro-inflammatory features. Our in vitro results indicate that, upon their engulfment, apoptotic hepatocytes laden with bile acids can serve as Trojan horses, delivering bile acids into efferocytic macrophages and thereby shaping macrophage function. This contrasts with the characteristics of macrophages that engulf apoptotic parenchymal cells lacking bile acids. Together, our findings delineate a system in which the content of the phagocytosed dying cells, specifically bile acid-laden hepatocytes, drives a pro-inflammatory program in the corresponding efferocytic macrophages, potentially contributing to chronic hepatic inflammation.