Proceedings of the National Academy of Sciences of the United States of America · 2019
Homozygous NLRP1 gain-of-function mutation in siblings with a syndromic form of recurrent respiratory papillomatosis.
Клинический случайСтатьяПоддержка NIH: внешняя программаПоддержка исследования: вне правительства СШАНазвание и аннотация: Перевод в очереди
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Drutman Scott B, Haerynck Filomeen, Zhong Franklin L, Hum David, Hernandez Nicholas J, Belkaya Serkan, Rapaport Franck, de Jong Sarah Jill, Creytens David, Tavernier Simon J, Bonte Katrien, De Schepper Sofie, van der Werff Ten Bosch Jutte, Lorenzo-Diaz Lazaro, Wullaert Andy, Bossuyt Xavier, Orth Gérard, Bonagura Vincent R, Béziat Vivien, Abel Laurent, Jouanguy Emmanuelle, Reversade Bruno, Casanova Jean-Laurent
Аннотация
Juvenile-onset recurrent respiratory papillomatosis (JRRP) is a rare and debilitating childhood disease that presents with recurrent growth of papillomas in the upper airway. Two common human papillomaviruses (HPVs), HPV-6 and -11, are implicated in most cases, but it is still not understood why only a small proportion of children develop JRRP following exposure to these common viruses. We report 2 siblings with a syndromic form of JRRP associated with mild dermatologic abnormalities. Whole-exome sequencing of the patients revealed a private homozygous mutation in NLRP1, encoding Nucleotide-Binding Domain Leucine-Rich Repeat Family Pyrin Domain-Containing 1. We find the NLRP1 mutant allele to be gain of function (GOF) for inflammasome activation, as demonstrated by the induction of inflammasome complex oligomerization and IL-1β secretion in an overexpression system. Moreover, patient-derived keratinocytes secrete elevated levels of IL-1β at baseline. Finally, both patients displayed elevated levels of inflammasome-induced cytokines in the serum. Six NLRP1 GOF mutations have previously been described to underlie 3 allelic Mendelian diseases with differing phenotypes and modes of inheritance. Our results demonstrate that an autosomal recessive, syndromic form of JRRP can be associated with an NLRP1 GOF mutation.